Exposure to stress in different periods of life represents a risk factor for the development
of psychiatric disorders. Here we used two different experimental paradigms, the prenatal
stress (PNS) model and the chronic mild stress (CMS) model to identify brain alterations
caused by exposure to stress early in life or at adulthood, and to investigate the
ability of the antipsychotic drug lurasidone to prevent or counteract the stress-induced
abnormalities.
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© 2020 Published by Elsevier Inc.
