Studies demonstrate that exposure to prenatal stress can have negative consequences
on neurodevelopment and has been linked with psychiatric disorders in the adult offspring.
Microglia have emerged as essential players in neurodevelopment, and whether microglia
shift to a pro-inflammatory phenotype with prenatal exposures, such as stress, is
an open question. In this study we address the contribution of maternal stress in
utero on microglia number and function.
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© 2020 Published by Elsevier Inc.
