Until recently, the goal of neuropsychological research on behaviorally defined disorders
has been to find the single underlying core deficit that provides a parsimonious,
causal explanation of the diversity of behavioral symptoms found in the disorder.
Both
Morton and Frith 1995
and
Pennington and Welsh 1995
explicated the logic of single-cognitive-deficit models of developmental disorders
like autism, dyslexia, and attention-deficit/hyperactivity disorder (ADHD). Although
testing such simple models was a reasonable initial strategy, it is becoming increasingly
clear that a single cognitive deficit will not suffice for any of these disorders
(
Pennington in press
).To read this article in full you will need to make a payment
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References
- By carrot or stick.Science. 2004; 306: 1940-1943
- Phonology, reading acquisition, and dyslexia.Psychol Rev. 1999; 106: 491-528
- Causal modeling.in: Cicchetti D. Cohen D.J. Developmental Psychopathology, Volume 1 Theory and Methods. John Wiley & Sons, New York1995: 357-390
- Causal heterogeneity in attention-deficit/hyperactivity disorder.Biol Psychiatry. 2005; 57: 1224-1230
- The Development of Psychopathology.Guilford, New York2002
Pennington BF (in press): A multiple-deficit model for understanding developmental disorders. Cognition.
- Neuropsychology and developmental psychopathology.in: Cicchetti D. Cohen D.J. Developmental Psychopathology, Volume 1 Theory and Methods. John Wiley & Sons, New York1995: 254-290
- Rodent models of attention-deficit/hyperactivity disorder.Biol Psychiatry. 2005; 57: 1239-1247
- Causal models of attention-deficit/hyperactivity disorder.Biol Psychiatry. 2005; 57: 1231-1238
- Modeling attention-deficit/hyperactivity disorder.Biol Psychiatry. 2005; 57: 1248-1255
Article info
Publication history
Published online: April 19, 2005
Identification
Copyright
© 2005 Society of Biological Psychiatry. Published by Elsevier Inc. All rights reserved.
